Dissociative stupor - Is it a disease or not? Psychologist's answer

Catatonia is a psychopathological syndrome with a predominance of psychomotor and autonomic disorders in the motor sphere, characterized by a clinical picture of agitation or stupor, which is unmotivated.

In the International Classification of Diseases (ICD-10), the disorder is considered within the framework of catatonic schizophrenia. This interpretation makes it difficult to diagnose the disorder.

Catatonic states of excitement and stupor can quickly replace each other. They arise autochthonously and are functional in nature. Catatonia may not be accompanied by stupefaction (lucid), or may be accompanied by oneiric stupefaction.

Catatonic stupor is characterized by increased muscle tone and immobility. Stupor can develop quickly or gradually. With gradual development, slowness, angular movements, prolonged freezing in one place (substupor), etc. initially appear. Over time, the severity of stupor increases.

Variants of motor stupor:

• Depressive;
• Catatonic;
• Hallucinatory;
• Apathetic;
• dissociative (“hysterical”);
• affective-shock.

Types of motor excitation:

• Manic;
• Anxious;
• Hallucinatory;
• Delusional;
• Affective-shock;
• Dissociative (“hysterical”);
• Against the background of confusion: delirium, twilight confusion.

Catatonic excitation develops suddenly, often rapidly moving from a state of stupor.

Catatonia is also characterized by other clinical manifestations:

Mutism is an unmotivated refusal to speak during normal functioning of the speech apparatus. Passive and active negativism is an unmotivated refusal to perform actions (for example, changing a position) or performing opposite actions. Motor and speech stereotypies - pretentiousness of movements and facial expressions. Echopraxia and echolalia are unmotivated repetition of the actions and phrases of others.

Causes

Specific reasons for the development of catatonia have not been established. The disorder may be initiated due to:

• Concomitant mental disorders (depression, bipolar disorder, schizophrenia, schizotypal disorder, acute psychosis);

• Substance and alcohol use;

• Neurological diseases (epilepsy, multiple sclerosis, Parkinson's disease, brain tumors, dementia);
• Metabolic and endocrine disorders (Cushing's syndrome, hyperthyroidism, Sheehan's syndrome);
• Autoimmune diseases (systemic lupus erythematosus, antiphospholipid syndrome), etc.

Genetic predisposition to certain mental disorders plays a great role. Some infectious diseases can also cause the development of catatonic stupor.

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• - Yes, because, it seems, it turns out... I even regret that I didn’t do this before. I mean prose. Sometimes I indulge in poems. When I needed to write about myself, I didn’t want to do a striptease in front of strangers. And then it clicked! I’ll write all this for my daughter! .Of course she knows a lot about me, but without details. Well, I’ll at least outline the main periods of my life. I got ready, wrote, and somehow it became easier, as if I had fulfilled my duty! And the shame in front of people melted away... And I realized that writing works if you do it honestly, sincerely, openly!

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Progress in three months

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Pathogenesis

There is no consensus on the pathogenesis of catatonia. Studies have shown that disruption of communication between cortical structures, the thalamus and the basal ganglia play an important role in the occurrence of catatonic symptoms, therefore, in addition to clinical assessment, when studying catatonia, it is worth taking into account the results of neuroimaging and neuropsychological examinations. The hypothesis is based on a lack of gamma-aminobutyric acid (GABA) in the basal ganglia. Normally, GABA reduces the intensity of emotional reactions, such as anger, fear or anxiety.

In modern literature, there are often cases of catatonia caused by abrupt withdrawal of drugs used to treat catatonic states and accompanying mental disorders. This therapy uses mechanisms to increase GABA activity in the basal ganglia, which has a positive therapeutic effect. With abrupt withdrawal of drugs, an increase in motor symptoms often occurs (the phenomenon of “rebound catatonia”).

It is possible that catatonia develops as a result of severe anxiety, in response to stress. Therefore, scientists suggest that catatonic states are directly related to affective and other mental disorders that are accompanied by symptoms of severe anxiety.

Stupor in the head: how to get rid of it - a simple debriefing

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Symptoms

There are many clinical signs of catatonic syndrome. Symptoms are often related to the motor area. With catatonic stupor, the patient becomes numb or completely immobile. Muscle tone is increased, especially in the head and shoulder girdle. The chewing muscles are toned, the lips are pulled forward (proboscis symptom), the person lies on his back, holding his head above the pillow (the “air cushion” symptom). Less severe conditions occur in the form of catalepsy. Muscle tone may alternate with complete relaxation, and epileptiform seizures are sometimes observed. The state of catatonic excitement is characterized by incoherent speech, absurd rhyming, and echolalia.

In severe cases, the attack of stupor with catalepsy is prolonged, mutism, lack of reaction of the pupils to intense painful stimuli (Bumke's symptom), and persistent insomnia are noted.

Stupor may be accompanied by oneiric stupefaction, delusions and hallucinations. Motor disturbances manifest themselves in combination with a feeling of stiffness of the body and the inability to move, with stupor, or a feeling of muscle looseness with motor excitement.

In catatonic states, the following symptoms may occur:

• catatonic stupor or substupor - complete or partial immobility, often in a sitting or standing position;
• catalepsy - a person remains in one position for a long time;
• waxy flexibility - long-term preservation of the position that another person gives the patient;
• negativism—resistance when another person tries to change the patient’s position;
• mutism – prolonged silence;
• stereotypy - repeated monotonous movements;
• catatonic agitation - increased disordered motor activity;
• echolalia/echopraxia - copying the words and movements of another person;
• pretentiousness of facial expressions and movements.

In addition to the main symptoms, manifestations of disruption of the autonomic nervous system may occur. With catatonia, there may be an increase in body temperature to subfebrile, a feeling of thirst, increased blood pressure, and tachycardia. Often patients refuse water and food.

The detailed picture of catatonic stupor and agitation does not differ in clinical manifestations in adolescents and adults. Oneiric catatonia often occurs against the background of the manic phase in schizoaffective psychosis; it is much less common in depression and attacks of fur-like schizophrenia, especially in adolescents. Hebephrenic syndrome is characteristic of adolescence and the malignant course of schizophrenia. As a rule, catatonic symptoms appear against the background of this syndrome.

Fibril catatonia develops with symptoms of oneiric, neurological and somatic disorders. A febrile attack is characterized by manic-delusional, hallucinatory and depressive syndromes. With more pronounced psychosis, fantastic delirium appears. The patient is in a state of agitation or stupor.

Body temperature rises sharply to febrile and higher. There is no relationship with somatic and infectious diseases. Blood pressure rises, tachycardia and increased sweating are present. The person is in an excited state and feels very thirsty. Excitement gives way to stupor or sub-stupor. General health worsens. The feeling of thirst and hunger disappears, the skin becomes dry, and swelling appears. If assistance is not provided in a timely manner, congestion in the lungs, bedsores and other complications may develop.

Most often, attacks of febrile catatonia are observed in people 41-50 years old. They are characterized by a severe course and a high risk of death.

With primary manifestations of catatonia, you should immediately consult a doctor. With timely diagnosis and effective treatment, the condition can be normalized without further development of complications.

Symptoms of stuporous catatonia

Stuporous catatonia is characterized by immobility during which people may assume rigid positions (stupor), an inability to speak (mutism), and a waxy flexibility in which they maintain positions after someone has placed them in them. Mutism may be partial, and they may repeat meaningless phrases or speak only to repeat what someone else says. People with dull catatonia may also exhibit stereotypic, repetitive movements (stereotypy). Excited catatonia is characterized by strange, non-goal-directedness and impulsivity.

Catatonia is a syndrome that can occur in a variety of mental disorders, including major depressive disorder, bipolar disorder, schizophrenia, schizoaffective disorder, schizophreniform disorder, brief psychotic disorder, and substance-induced psychotic disorder. It manifests itself as Kahlbaum's syndrome (immobile catatonia), malignant catatonia (neuroleptic malignant syndrome, toxic serotonin syndrome) and excited forms (delusional mania, catatonic agitation, oneirophrenia). He has also been assessed for autism spectrum disorder.

Classification

Catatonic states are classified based on the severity of clinical manifestations and the nature of the course of the disorder. Currently, there are two types of catatonia: benign and malignant. Malignant catatonia is the most severe form of the disorder and poses a threat to the patient’s life (“lethal” or “febrile” catatonia).

Febrile catatonia is an acute mental disorder accompanied by a deterioration in general physical condition: increased body temperature, somatovegetative disturbances, homeostasis disorders, deterioration in the functional ability of body systems. The condition develops without connection with somatic diseases. With such a course of the disorder, there is a possible risk of developing cerebral edema and multiple organ failure

Catatonia that occurs at the onset of the disease is considered primary (often observed in schizophrenia). If catatonic manifestations develop against the background of a prolonged course of the disease, catatonia is secondary.

Often in psychiatry, a classification of catatonia is used, based on the pattern of the underlying disorder and the predominance of catatonia syndrome over other symptoms. According to these signs, minor and extensive catatonia are distinguished.

Mild (small) catatonia

. is a reduced version of the catatonic syndrome, in which some simple manifestations are present. Negativism is not clearly expressed, substuporous states, short-term stops of movements and speech, episodic echolalia, isolated grimaces and unexpected actions are noted. The patient enters into dialogue only under duress, is reluctant to answer questions, his speech is mannered, and he avoids visual and tactile contact with the interlocutor. This condition is most often observed in developing schizophrenia.

According to the degree of severity, catatonic excitation is:

• ecstatic - patients are mobile, talk a lot;
• hebephrenic - ridiculous, inappropriate jokes, unmotivated and unreasonable gaiety predominate;
• impulsive - unexpected actions, often destructive and aggressive;
• silent - motor agitation with aggression and destructive actions.

Silent (mute) catatonic agitation

. The person makes meaningless, chaotic movements and resists other people’s attempts to help. During this period of arousal, there is a risk of harm to yourself and others.

Impulsive catatonic agitation

. Development is fast. A person copies the movements and phrases of nearby people. Speech is limited to a chaotic set of phrases. There is a tendency towards cruelty and destructive actions, and a high risk of self-injury.

Ecstatic

. Symptoms increase gradually. At the beginning, there is little motor and speech activity. The nature of the speech is mannered, echolalia (copying other people's words) is possible, and laughter often occurs for no apparent reason.

Degrees of severity of catatonic stupor:

Cataleptic

(with waxy flexibility). Muscle tone is increased, a person can maintain an uncomfortable position for a long time. Manifested by symptoms: catalepsy, waxy flexibility (maintains a position devoted to another person), “air cushion” (characteristic position on the back with the head raised above the pillow).

With negativism

. More pronounced muscle tension. There is sharp opposition to passive movements and attempts by other people to change the patient’s posture. Mutism is often observed.

Rigid

. Muscle tone is very pronounced. Due to the predominance of flexor muscle tone, the person is in the fetal position (knees brought to the stomach, hands pressed to the chest). Trying passive movements increases muscle tension. Muscle tone decreases during deep sleep.

Catatonic states are divided into two types - oneiric and lucid, depending on the presence of clouding of consciousness. With oneiric clouding of consciousness, ecstatic, impulsive, hebephrenic excitement, stupor with waxy flexibility and substuporous states develop. With lucid catatonia, stupor with negativism often develops.

Oneiric catatonia

combines a state of stupor and a dream-like state with vivid scene-like hallucinations, which the patient can talk about after emerging from the stupor. During stupor, the patient is most often disoriented and unable to perceive surrounding events; perception occurs in a distorted form with dream-like fantasies.

Lucid catatonia

represents a stupor without clouding of consciousness. The perception of what is happening around is not distorted. The patient is normally oriented in time and space, remembers the events that occurred while in a stuporous state. Sometimes partial amnesia occurs regarding one's own movements and behavior. The patient may not remember his own impulsive actions, prolonged immobility, etc.

Taking into account diagnostic approaches, catatonia is classified according to provoking factors:

Catatonia developed against the background of concomitant mental disorders. Most often, catatonic schizophrenia is considered (if motor disorders are the main clinical symptoms of the disorder), affective disorders with catatonic symptoms, and others.

Catatonia due to somatic illness. Catatonic syndrome may indicate neurological, infectious and other diseases with metabolic disorders.

Unspecified catatonia. Catatonic states without correlation with other mental and somatic disorders.

Types of stupor

There are several types of stupor: negativistic, depressive, apathetic and catatonic, as well as a stuporous state with waxy flexibility or muscle numbness.

Negativistic stupor is expressed in mutism and absolute immobility, but at the same time, any action aimed at changing the patient’s posture provokes sharp opposition and resistance. It is not easy to lift a sick individual out of bed, but then, having lifted him, it is impossible to put him back down. Often active resistance is added to passive resistance. For example, if a doctor extends his hand to a patient, he, in turn, hides his hand behind his back, when asked to open his eyes, he closes his eyes, etc.

A depressive stuporous state is characterized by almost complete immobility along with a depressed facial expression and a pained grimace. When you manage to establish contact with them, you can get a monosyllabic response.

A person's depressive stupor may suddenly give way to an excited state, in which patients jump up and hurt themselves, may injure themselves, or roll on the floor howling (melancholic raptus). In severe depression of an endogenous nature, depressive stupor may occur.

Patients suffering from apathetic stupor usually lie on their backs. They also do not react to what is happening around them, and their muscle tone is reduced. They answer questions in monosyllables and with a long delay. However, during interactions with relatives, an adequate emotional reaction is observed. There are sleep disorders and appetite disturbances. They are often untidy in bed. Catatonic stupor is a kind of freezing in fear, numbness in fear and helplessness along with severe suffering of the inner “I”. Patients with catatonia sometimes do not understand whether they are still alive, whether they are capable of performing actions, and are not confident in the integrity of their own personality. Therefore, everything that can lead to the reconstruction of the authenticity of the self-experience will play a therapeutic role for the patient.

For example, with the loss of self-identity, sometimes it is enough just to call by name to improve the patient’s condition. How to get out of the stupor? In severe cases of the disease, a purely verbal therapeutic approach is often insufficient. Other types of catatonic stupor appear when overloaded with delusional experiences, for example, when the individual is in a state of ecstasy.

In a stuporous state with waxy flexibility, in addition to mutism and immobility, the patient holds the crouched position for a long time. For example, he freezes with his hand raised or freezes in an awkward position. The presence of Pavlov's symptom is often noted, which consists in the absence of a reaction in patients to question phrases asked in a normal voice, but at the same time responds to a whisper. At night, sick individuals can walk, sometimes eat and interact with the environment.

A stuporous state with muscle numbness represents being in the fetal position. In such patients, the muscles are tense, the eyes are closed, and the lips are extended forward. Often, individuals suffering from this type of stupor must be fed through a tube because they refuse to eat. Often, doctors perform amytalcaffeine disinhibition, and after the muscle numbness weakens or disappears, they feed the patients.

Complications

The development of complications is often associated with late diagnosis and incorrect approach to treating the disorder.

When treating people with severe forms of catatonia, difficulties may arise in the daily care of patients, as a result of which complications may develop:

• aspiration pneumonia - occurs due to the difficulty of eating in patients with mutism and the risk of aspiration;
• risk of deep vein thrombosis and pulmonary embolism due to immobility in people with catatonia;
• metabolic disorders associated with dehydration and exhaustion of the body - patients with catatonia may not eat or drink for a long time;
• disorders of the gastrointestinal tract (constipation, intestinal obstruction, etc.)
• urinary retention or incontinence;
• neuromuscular complications (flexion contracture, nerve palsy)
• formation of bedsores.

Also, in a state of catatonic excitement, the patient poses a danger to himself and others. The sudden onset of an attack of stupor can lead to an accident, overtaking a person driving a car, while crossing the road, at work, etc.

Complications of severe forms of catatonia are difficult to treat due to the difficulties in contact between the patient and the doctor.

What is dissociative stupor

Dissociative dementia (dissociative stupor) is a movement disorder caused by trauma.
It is characterized by mutism and complete or almost complete immobility, but does not cause any physical or mental impairment. Develops as a result of excessive acute stress, severe social or interpersonal problems. The duration usually varies from a few minutes to several hours. The diagnosis is made based on clinical symptoms and data on the traumatic event. During the diagnostic process, other psychiatric and somatic pathologies are excluded. Treatment consists of psychotherapy and pharmacological therapy.

ICD-10

Surely, you have more than once been in a state of some kind of stupor, when it is impossible to make a decision, comprehend the problem, or draw a conclusion. Is this condition a disease? No. But it is dissociative stupor, which is caused by trauma, mental dysfunction, that is a disease.

In the international classifier ICD-10, dissociative stupor is allocated to a separate category of diseases - F44.2 Dissociative stupor.

Dissociative stupor is a disease caused by a traumatic situation or stress.

Level of consciousness during stupor

As for consciousness in this situation, the patient is quite vague and cannot answer any questions; reactions are short-lived, even to strong stimuli.

Biological basis

Man is not as far from animals as we would like to think. This condition also occurs in the animal world, and is not so rare. Think of a rabbit, stunned, waiting to be swallowed by a boa constrictor. Or a chicken that freezes when its head is placed under its wing.

In the animal kingdom, stupor is a “mercy”: it allows you to die painlessly, rendering the victim unconscious and leaving him with no choice but to surrender. In this state, the pain subsides and merciful insensibility sets in.

Psychological trauma can be unbearable for a person, the situation can be perceived as hopeless and unpromising, and this is an important element in causing stupor.

Diagnostics

When diagnosing catatonic states, it is important to correctly assess the patient’s clinical condition. When collecting anamnesis and examination, it often turns out that the person used psychoactive substances, which most likely contributed to the development of catatonic syndrome. It is worth considering the likelihood of developing symptoms of catatonia during treatment with psychotropic drugs for concomitant diseases.

Psychometric scales are used to diagnose the disorder. The most commonly used is the Bush-Francis Catatonic Disorder Scale. It allows you to determine the presence and severity of catatonic symptoms over a certain period of time. Other rating scales are also used.

Electroencephalography (EEG), CT, MRI and neuropsychological examinations allow us to suggest the localization of disorders in the brain and diagnose the functional state of higher mental functions.

Differential diagnosis is carried out with other mental and somatic disorders accompanied by motor disorders. The most clinically similar to the catatonic syndrome is Parkinson's disease, but the disease is accompanied by a number of specific symptoms that make it possible to differentiate it from catatonia (tremor, apraxia of walking).

Catatonia is differentiated from schizophrenic catatonia, acute psychoses with motor agitation, conversion disorders and other conditions with severe motor retardation or agitation, and neurological symptoms.

Causes of stupor

Stupor is a psychopathological disorder that manifests itself in the form of suppression of various mental operations, primarily motor skills, mental activity and speech. Patients who find themselves in this state are characterized by immobility. Left to themselves, sick individuals remain in one position for a long time. They either may not respond to question phrases at all, or they answer, but after a pause, at a slow pace, with interjections, individual words, or only occasionally in short phrases.

In some cases, the disease can occur in combination with a variety of psychopathological symptoms, such as delirium, hallucinosis, confusion, and altered affect. In other situations, more rare, the stuporous state is limited solely to motor immobility and speech retardation. In other words, this state is also called “empty” stupor.

Stupor, which is accompanied by confusion, is called receptor stupor. A stuporous state observed in conditions of clear consciousness is called lucid or effector.

The main factors provoking the occurrence of a stuporous state include severe psychotraumatic events, stressful situations, mental disorders, negatively emotional emotional situations, organically determined lesions of brain structures, various bruises or concussions, intoxication, and infectious disease. However, to this day it cannot be said with one hundred percent probability that the listed list of reasons is complete.

World-renowned specialists in the field of psychiatry enter into discussions about the possible causes provoking the development of the disease. Thus, among the numerous assumptions, there are several that are most characteristic of the formation and formation of an immobile type of catatonic stupor. Deficiency of gamma-aminobutyric acid in the brain, which is its key inhibitory neurotransmitter. A lack of this acid can cause disorders of the musculoskeletal system. And this is the main symptom of catatonia. Catatonic stupor can occur due to an unexpected stop in the body's production of dopamine.

In 2004, experts began to consider the formation of catatonic syndrome as a genetic reaction that occurs in situations of stress or life-threatening circumstances in animals before meeting a predator. The whole body is paralyzed due to fear, as a result of which the animal’s body is reconfigured for imminent death. This reaction of fear on a subconscious level has been preserved in humans and to this day manifests itself during exacerbations of psychosomatic diseases or intense attacks of schizophrenia.

Catatonic stupor, according to this assumption, is expressed in the characteristic reaction of individuals to the inevitable death that has haunted him since the onset of the disease. Thus, the listed hypotheses determine the emergence of catatonic syndrome as a consequence of the presence of schizophrenia and other diseases of a psychosomatic nature.

Forecast

Mild forms of catatonia usually do not have a strong impact on the patient’s further social adaptation and respond well to treatment. Treatment is carried out in a psychiatric hospital. In case of severe somatic diseases, hospitalization is carried out in a specialized department (neurological, oncological). A poor prognosis is associated with catatonia of a malignant course, due to the risk of death, and the presence of catatonia in children, adolescents and the elderly with schizophrenia. Such people require constant psychiatric treatment. Relapses of catatonic states most often occur with the idiopathic version of the disorder or in the presence of concomitant affective disorders. If a person has renal failure, Parkinson's disease, or alcohol addiction, the dysfunction of the brain is chronic, which can also lead to relapses.

In most cases, the prognosis of catatonic syndrome depends on the course of the underlying disease and timely treatment.

Causes of development of dissociative stupor

The source is always a traumatic situation, but the nature, duration and objective significance of these situations can vary greatly. Clinically significant stupor occurs most often during large, destructive events that pose an immediate threat to human life. These include floods, earthquakes, hurricanes, house collapses, industrial accidents, train crashes, wars, etc.

In addition to the threat to life, the high likelihood of developing dissociative states in such situations is due to the peculiarities of perceiving oneself as small, helpless and insignificant compared to the forces of nature or other similar phenomena (insignificance in the face of fate). In addition, stupor can be provoked by tragic events that threaten a person: car accidents, criminal events (especially violent ones), etc.

The development of a state of stupor is sometimes provoked by situations that are not life-threatening, but extremely important for the patient - the death of a loved one, separation from loved ones, bankruptcy or staff reduction. Brief dissociative stupor without clinical significance can occur with any sufficiently severe acute stress, such as the threat of being attacked by a large dog or the threat of a car accident. In children, such conditions can occur during exams, conflicts with peers and other stressful situations.

The likelihood, depth and duration of stupor depend on three factors: the severity of the threat (including subjective assessment), the type and characteristics of the reactivity of the patient’s nervous system, as well as the psychophysical state of the patient at the time of the traumatic situation. Assessing the seriousness of a threat and psychological readiness for sudden stress is determined by professional and life experience (children often perceive threats more seriously than adults, people in “quiet” professions more seriously than rescuers, military personnel or emergency doctors).

Stupor often occurs in people prone to “frozen”, unstable behavior in unforeseen circumstances. Physical fatigue or overwork due to too much strenuous work, lack of sleep, or acute or chronic medical illness increases the risk of developing the disorder. Psychological exhaustion due to constant tension or internal conflicts plays an unfavorable role. Traumatic childhood experiences are also important.

Treatment in Re-Alt

When treating catatonia that has developed against the background of another mental or physical disorder, therapy is aimed mainly at the underlying disease. Therefore, it is very important for the effectiveness of treatment to conduct a thorough diagnosis. At the Re-Alt mental health center, specialists conduct a detailed analysis of each clinical case and assess the patient’s mental and somatic status in order to individually select treatment tactics.

The most effective method of treating catatonia is the use of drugs from the group of benzodiazepine tranquilizers. Pharmacotherapy helps reduce muscle tone and eliminate other manifestations of catatonia. Treatment uses drugs with sedative, anticonvulsant effects, and muscle relaxants.

If drug treatment is ineffective, electroconvulsive therapy may be prescribed.

Febrile catatonia is a medical emergency. Treatment is carried out inpatiently. Measures are prescribed to maintain and normalize the functioning of all body systems. Antibacterial therapy is prescribed to prevent complications. After stabilization of the condition, electroconvulsive therapy is performed

Treatment of stupor

Many people are concerned about the question: “how to get out of the stupor”? Naturally, only specialists - psychotherapists and psychologists - can help with this. However, you should still know how to help a loved one or someone around you if signs become noticeable that the subject is about to fall into a stupor or has already entered such a state and needs help.

So, first of all, massage of special points located exactly in the middle above the pupils, equidistant from the eyebrow arches and hairline, will help relieve tension. These points should be massaged using the pads of the index finger and thumb. In addition, it is recommended to try to provoke strong emotions in an individual in a stuporous state, no matter positive or negative (preferably negative). For example, you can slap someone in the face.

It can help to get out of the stupor by bending the individual’s fingers and pressing them forcefully against the palms, while the thumbs remain straight. So, the answer to the question: “how to get out of a stupor” is hidden in the emotional shake-up of the body and the synchronization of the breathing of the sufferer with the subject helping him. For this purpose, you can put your hand on the chest of an individual who has fallen into a stupor and adjust to his breathing pace.

In case of stupor, emergency care is limited to ensuring the safety of subjects and preventing dangerous actions on their part. For example, in a catatonic stuporous state, emergency assistance will consist of readiness to stop unexpected impulsive agitation.

In case of depressive stupor - preventing the possibility of unexpected development of depressive agitation with a focus on suicide, as well as eliminating food refusal. In addition, you need to take into account that stupor can suddenly give way to excitement.

Treatment often occurs in an inpatient setting. Barbamyl-caffeine disinhibition is used. Thanks to which it is possible to detect the characteristics of the patient’s experiences and anxieties, which helps to determine the nature of the stuporous state. This disinhibition is also a therapeutic method that helps with persistent food refusal.

A stuporous state that occurs against the background of severe somatic illnesses requires treatment of the underlying disease.

For stupor accompanied by hallucinations and delusions, Stelazine and Trisedal are used as well as in the treatment of hallucinatory and delusional states. In case of a depressive stuporous state, disinhibition is also carried out and Melipramin is used up to 300 mg per day orally or intramuscularly. For a psychogenic stuporous state - Diazepam up to 30 mg per day orally or intramuscularly, Elennium or Phenazepam.